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In Gones, Italy, a recovered COVID-19 patient undergoes a center service as a test.
Science COVID-19 reports are supported by the Pulitzer Center and the Heising-Simons Foundation.
Fears that COVID-19 could cause heart inflammation called myocarditis are higher, as doctors report seeing other healthy people in the past whose experience with COVID-19 is accompanied by a central failure induced by myocarditis. COVID-19 symptoms in April, adding loss of smell and mild shortness of breath. A month later, he became seriously ill with severe myocarditis. “I’m grateful to be alive,” says Kashem, who spent more than 2 weeks in an intensive care unit Why did this happen?
But some cases result in arrhythmias and the deteriorated center serves as or rarely the desire for a center transplant. Because millions of others now get coronavirus, even a small proportion suffering from severe myocarditis would be severe for many other people. is there going to be an increase in the number of patients with center failure as a result?”Peter Liu, cardiologist and clinical director of the Heart Institute at the University of Ottawa, asks.
It is unclear whether SARS-CoV-2, the virus that causes COVID-19, induces central damage, adding myocarditis, more or more severely than other viruses, because SARS-CoV-2 can cause an intense immune reaction throughout the body. Another concept suggests that patients with COVID-19 may also be prone to disease because the virus enters cells through binding to the angiotensin 2 conversion enzyme (ACE2) receptor, which is discovered at the center. But researchers warn that they are opposed to exceeding the data. “It’s a smart hypothesis, but it’s not a proven hypothesis,” said Leslie Cooper, cardiologist at the Mayo Clinic in Jacksonville, Florida, about ACE2.
One of the reasons it is difficult to say whether COVID-19 poses a specific threat of myocarditis is uncertainty about its prevalence after other infections. Echocardiogram studies after some influenza outbreaks recommend that up to 10% of influenza patients have brief central defects, Liu But these studies are rare. “We don’t test patients after they’ve contracted the flu,” says Valentina Pantmann, cardiologist at Frankfurt University Hospital.
She fed considerations about myocarditis when she did just that with COVID-19 patients. His team used MRI to scan the hearts of a hundred PATIENTS with COVID-19 on average 71 days after their positive test. of which they gave the impression of having active inflammation. Most also described persistent symptoms, such as fatigue and mild shortness of air, which led to the question of whether heart inflammation might be responsible.
Although the paintings of P-ntmann and his colleagues, published in July in JAMA Cardiology, have been alarmingly news, many researchers say it wants to be reproduced. seek medical attention, but are involved in an avalanche of other healthy people who are asking for evaluations from the center. “Here’s the good news: let’s find out” how it’s more likely to have an injury downtown, says Matthew Martinez, director of sports cardiology at Morristown Medical Center.
Due to the physical demands of the sport, the team’s doctors deserve to be alert to myocarditis. An article in JAMA Cardiology last week reported a 26-athlete exam at Ohio State University after COVID-19; 4 had evolved with myocarditis. Professional sports leagues also analyze the hearts of inflamed athletes with SARS-CoV-2. Martinez, who is helping coordinate studies for the National Basketball Association and Major League Soccer, predicts a stream of athlete knowledge over the coming months. “Those of us in this area are willing to lose a Saturday or Sunday to get there. “
However, he says that while researchers may explain the average duration of myocarditis and its dangers to a young athlete, those can be very different for a 50-year-old man with obesity or high blood pressure, especially if he is in poor health. coVID-19 enough to hospitalize. ” In those people, I’m going to be more careful” and detect injuries at the center,” he said.
Others are looking for clues as to how the COVID-19 can damage the center, possibly involving tactics to save it. “SARS-CoV-2 requires situations that require its immune formula in an unconventional way,” Liu says. tissue after COVID-19 revealed inflammation of the center’s blood vessels instead of its muscle cells, the site of inflammation caused by other infections. Another post-mortem examination revealed the scattered death of the center’s cells, however, the authors noted that the wound mechanism was “Much debate has been discussed about whether it is myocarditis,” as defined, Liu says. In any case, he and others expect clinical trials to check whether life-saving strategies, such as taking beta blockers, can also save you central failure in a user reported as the main threat after COVID-19.
While Mohiddin volunteered for a survivor’s exam, he also led one: a trial that aims to recruit 140 others while hospitalized with or after COVID-19, 20 with and the others with severe myocarditis. it will look for abnormal degrees of T cells in other people’s blood with myocarditis, which can help if the immune formula causes central damage and how it does.
Although COVID-19 rarely causes severe myocarditis, speculation is that mild cases can also increase the threat of central disease years later. Scar tissue would possibly form as myocarditis heals, and previous paints have shown that residual heart inflammation suggests a deterioration in the center As cardiologists, “we are in the box to identify asymptomatic threat factors,” says Mohiddin. “It’s not hard to believe that in the long run doctors will ask a new patient, “Have you had a COVID?”»
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